<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1688-4809</journal-id>
<journal-title><![CDATA[Veterinaria (Montevideo)]]></journal-title>
<abbrev-journal-title><![CDATA[Veterinaria (Montev.)]]></abbrev-journal-title>
<issn>1688-4809</issn>
<publisher>
<publisher-name><![CDATA[Sociedad de Medicina Veterinaria del Uruguay]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1688-48092025000201204</article-id>
<article-id pub-id-type="doi">10.29155/vet.61.224.6</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[Evaluation of potential kidney damage associated with the use of phenobarbital and potassium bromide in epileptic canines using biomarkers predicting early damage]]></article-title>
<article-title xml:lang="es"><![CDATA[Evaluación del potencial daño renal asociado al uso de fenobarbital y bromuro de potasio en caninos epilépticos mediante el empleo de biomarcadores predictores de daño precoz]]></article-title>
<article-title xml:lang="pt"><![CDATA[Avaliação do potencial de dano renal associado ao uso de fenobarbital e brometo de potássio em cães epilépticos mediante o uso de biomarcadores preditivos de lesão renal precoce]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Brighenti]]></surname>
<given-names><![CDATA[Clarisa]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Capelli]]></surname>
<given-names><![CDATA[Alejandra]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Norbis]]></surname>
<given-names><![CDATA[Walter]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Della Cella]]></surname>
<given-names><![CDATA[Claudia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Delucchi]]></surname>
<given-names><![CDATA[Luis]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Fagúndez]]></surname>
<given-names><![CDATA[Catherine]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de la República Facultad de Veterinaria Departamento de Clínica y Hospital Veterinario]]></institution>
<addr-line><![CDATA[Montevideo ]]></addr-line>
<country>Uruguay</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de la República, Facultad de Veterinaria Departamento de Patobiología]]></institution>
<addr-line><![CDATA[Montevideo ]]></addr-line>
<country>Uruguay</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad de la República Facultad de Ciencias Departamento de Biología Animal]]></institution>
<addr-line><![CDATA[Montevideo ]]></addr-line>
<country>Uruguay</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2025</year>
</pub-date>
<volume>61</volume>
<numero>224</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.edu.uy/scielo.php?script=sci_arttext&amp;pid=S1688-48092025000201204&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.edu.uy/scielo.php?script=sci_abstract&amp;pid=S1688-48092025000201204&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.edu.uy/scielo.php?script=sci_pdf&amp;pid=S1688-48092025000201204&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Epilepsy is a neurological disease present in a wide range of mammals, including humans and domestic canines and felines. The most commonly used pharmacological treatments for canines are phenobarbital and potassium bromide (KBr). However, both of these medications are partially eliminated through the kidneys, which could potentially cause damage at this level. Current diagnostic methods for kidney damage typically identify issues only at advanced stages, highlighting the need for early detection techniques. Thus, this study aims to evaluate early renal alterations in canines diagnosed with idiopathic epilepsy and treated with phenobarbital and/or KBr, using specific biomarkers such as kidney injury molecule 1 (KIM-1) and neutrophil gelatinase-associated lipocalin (NGAL), alongside non-specific biomarkers such as C-reactive protein (C-RP). Data from 39 canines treated at the Veterinary Hospital Centre (Udelar) and private clinics in Montevideo, Uruguay, were analysed. Subjects were categorised into two groups: a control group (healthy animals) and a group of subjects previously diagnosed with idiopathic epilepsy. The latter group was subdivided into three additional groups: one treated with phenobarbital, one treated with phenobarbital/KBr, and one treated with KBr alone. Laboratory results were evaluated for new biomarkers and conventional analytes. No alterations were found in laboratory tests for liver function, profiles, or urinalysis among the 30 canines subjected to the different treatments. Regarding specific biomarkers, no statistically significant differences (p &gt; 0.05) were observed among the groups. However, alterations in C-RP were found in the group treated only with KBr. Under the conditions of the trial, no significant changes were detected in urinalysis or renal profiles associated with the administration of phenobarbital and/or potassium bromide in canines with idiopathic epilepsy.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La epilepsia es una enfermedad neurológica presente en casi todos los mamíferos, incluidos humanos, caninos y felinos. Los tratamientos farmacológicos más utilizados para los caninos son el fenobarbital y el bromuro de potasio (KBr). Sin embargo, ambos medicamentos son eliminados en parte por la vía renal, lo que potencialmente podría provocar daño a este nivel. Los métodos de diagnóstico convencionales utilizados para detectar daño renal identifican problemas en etapas avanzadas, lo que remarca la necesidad de técnicas de detección temprana. Este estudio tiene como objetivo evaluar las alteraciones renales tempranas en caninos diagnosticados con epilepsia idiopática y tratados con fenobarbital y/o KBr, utilizando biomarcadores específicos, como Kidney Injury Molecule-1 (KIM-1) y Neutrophil Gelatinase-Associated Lipocalin (NGAL), junto con biomarcadores no específicos, como la proteína C reactiva (PCR). Se analizaron datos de treinta y nueve caninos atendidos en el Centro Hospitalario Veterinario (Udelar) y clínicas privadas de Montevideo, Uruguay. Los animales se clasificaron en dos grandes grupos: uno de control (animales sanos) y otro de animales previamente diagnosticados con epilepsia idiopática. Este último se subdividió en tres grupos: tratamiento con fenobarbital, tratamiento con fenobarbital/KBr y tratamiento con KBr. Se evaluaron los resultados de laboratorio de los nuevos biomarcadores y de analitos convencionales. No se encontraron alteraciones en pruebas de laboratorio de función hepática, perfiles renal y análisis de orina para los treinta caninos sometidos a los diferentes tratamientos. En cuanto a los biomarcadores específicos, no se observaron diferencias estadísticamente significativas (p &gt; 0,05) entre los grupos. Sin embargo, se encontraron alteraciones en la proteína C reactiva en el grupo tratado solo con KBr. En resumen, bajo las condiciones del ensayo, no se detectaron cambios significativos en el análisis de orina ni en los perfiles renales asociados con la administración de fenobarbital y/o bromuro de potasio en caninos con epilepsia idiopática.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo A epilepsia é uma doença neurológica presente em uma ampla gama de mamíferos, incluindo humanos, caninos e felinos domésticos. Os tratamentos farmacologicos mais comumente utilizados para caninos são o fenobarbital e brometo de potássio (KBr). No entanto, ambos os medicamentos são parcialmente eliminados pelos rins, o que pode causar danos nesse nível. Os métodos atuais de diagnóstico de danos renais normalmente identificam problemas apenas em estágios avançados, destacando a necessidade de técnicas de detecção precoce. Assim, este estudo tem como objetivo avaliar alterações renais precoces em caninos com diagnóstico de epilepsia idiopática e tratados com fenobarbital e/ou KBr, utilizando biomarcadores específicos como Kidney Injury Molecule-1 (KIM-1) e Neutrophil Gelatinase-Associated Lipocalin (NGAL), juntamente com biomarcadores inespecíficos, como a proteína C reativa (PCR). Foram analisados dados de 39 caninos atendidos no Centro Hospitalar Veterinário (Udelar) e clínicas privadas de Montevidéu, Uruguai. Os indivíduos foram categorizados em dois grupos, um grupo controle (animais saudáveis) e um grupo de indivíduos previamente diagnosticados com epilepsia idiopática. Este último grupo foi subdividido em outros três grupos, um grupo tratado com fenobarbital, um grupo tratado com fenobarbital/KBr e um grupo tratado apenas com KBr. Os resultados laboratoriais de novos biomarcadores e analitos convencionais foram avaliados. Não foram encontradas alterações nos exames laboratoriais de função hepática, perfis ou urinálise entre os trinta caninos submetidos aos diferentes tratamentos. Em relação aos biomarcadores específicos, não foram observadas diferenças estatisticamente significativas (p &gt; 0,05) entre os grupos. Porém, foram encontradas alterações na PCR no grupo tratado apenas com KBr. Em resumo, nas condições do ensaio, não foram detectadas alterações significativas no exame de urina ou nos perfis renais associados à administração de fenobarbital e/ou brometo de potássio em caninos com epilepsia idiopática.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Early diagnostic methods]]></kwd>
<kwd lng="en"><![CDATA[New kidney biomarkers]]></kwd>
<kwd lng="en"><![CDATA[Antiepileptic treatments.]]></kwd>
<kwd lng="es"><![CDATA[Métodos de diagnóstico temprano]]></kwd>
<kwd lng="es"><![CDATA[Nuevos biomarcadores renales]]></kwd>
<kwd lng="es"><![CDATA[Tratamientos antiepilépticos.]]></kwd>
<kwd lng="pt"><![CDATA[Métodos de diagnóstico precoce]]></kwd>
<kwd lng="pt"><![CDATA[Biomarcadores de notícias renais]]></kwd>
<kwd lng="pt"><![CDATA[Tratamentos antiepilépticos.]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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